Effect of lack of Interleukin-4, Interleukin-12, Interleukin-18, or the Interferon-gamma receptor on virus replication, cytokine response, and lung pathology during respiratory syncytial virus infection in mice.

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作者： Boelen A ^[1] ; Kwakkel J ^[2] ; Barends M ; de Rond L ; Dormans J ; Kimman T

作者单位： .cbiba-u.ac.jp ^[1] Research Laboratory for Infectious Diseases, National Institute of Public Health and the Environment, Bilthoven, The Netherlands. ^[2]

关键词 Cytokines Lung Mice Knockout Respiratory Syncytial Virus Infections 细胞活素类肺小鼠基因摧毁呼吸道合胞体病毒感染呼吸道合胞病毒

发布时间 2013-11-20

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Journal of Medical Virology

2002年66卷4期

552-560页

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Effect of lack of Interleukin-4, Interleukin-12, Interleukin-18, or the Interferon-gamma receptor on virus replication, cytokine response, and lung pathology during respiratory syncytial virus infection in mice.

Journal of Medical Virology

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Journal of Medical Virology

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Effect of lack of Interleukin-4, Interleukin-12, Interleukin-18, or the Interferon-gamma receptor on virus replication, cytokine response, and lung pathology during respiratory syncytial virus infection in mice.

Journal of Medical Virology

相似文献

Journal of Medical Virology

相关期刊

检索历史清除