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Coexpression of heat-shock protein 60 and intercellular-adhesion molecule-1 is related to increased adhesion of monocytes and T cells to aortic endothelium of rats in response to endotoxin.

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第一作者： Seitz CS

作者： Seitz CS ^[1] ; Kleindienst R ^[2] ; Xu Q ; Wick G

作者单位： Institute for General and Experimental Pathology, Medical School, University of Innsbruck, Austria. ^[1] dfar3.dsfarm.unipd.it ^[2]

关键词 Chaperonin 60 Endothelium Vascular Intercellular Adhesion Molecule-1 监控蛋白60 内皮血管胞间粘附分子1 脂多糖类单核细胞 T淋巴细胞

发布时间 2013-11-20

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Laboratory investigation

1996年74卷1期

241-252页

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Laboratory investigation

1996年74卷1期

241-252页

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Coexpression of heat-shock protein 60 and intercellular-adhesion molecule-1 is related to increased adhesion of monocytes and T cells to aortic endothelium of rats in response to endotoxin.

Laboratory investigation

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Laboratory investigation

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Coexpression of heat-shock protein 60 and intercellular-adhesion molecule-1 is related to increased adhesion of monocytes and T cells to aortic endothelium of rats in response to endotoxin.

Laboratory investigation

相似文献

同项目论文

Laboratory investigation

相关期刊

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