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Deletion of the last five C-terminal amino acid residues of connexin43 leads to lethal ventricular arrhythmias in mice without affecting coupling via gap junction channels

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第一作者: Lübkemeier,I.
作者单位: Institute of Medicine-Cardiology, University of Bonn, Bonn, Germany [1] Center for Sepsis Control and Care (CSCC), Jena University Hospital, Jena, Germany [2] Dominick P. Purpura Department of Neuroscience, Albert Einstein College of Medicine, New York, NY [3] Department of Pathology, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul [4] Department of Biological Sciences, Rutgers University, Newark, NJ, United States [5] Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany [6] Institute of Physiology i, Life and Brain Center, University of Bonn, Bonn, Germany [7] Life and Medical Sciences (LIMES) Institute, Molecular Genetics, University of Bonn, Carl-Troll-Str [8] Institute of Cellular Neurosciences, University of Bonn, Bonn, Germany [9] Departments of Internal Medicine and Physiology, Dorothy M. Davis Heart and Lung Research Institute [10] Leon H. Charney Division of Cardiology, New York University Medical School, New York, NY, United [11]
发布时间 2013-11-20
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Basic Research in Cardiology: Official Journal of the German Association of Cardiovascular Research

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