Fanning the Flames of Endoplasmic Reticulum (ER) Stress: Can Sphingolipid Metabolism Be Targeted to Enhance ER Stress-Associated Immunogenic Cell Death in Cancer?

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作者： Hengst, Jeremy A. ^[1] ; Nduwumwami, Asvelt J. ^[2] ; Sharma, Arati ; Yun, Jong K.

作者单位： Pennsylvania Commonwealth System of Higher Education (PCSHE),Penn State College of Medicine ^[1] National Institutes of Health (NIH) - USA,Natl Ctr Adv Translat Sci ^[2]

关键词 SPHINGOSINE KINASE 1 CERAMIDE SYNTHASE 6 ACID CERAMIDASE DIHYDROCERAMIDE DESATURASE CALRETICULIN EXPOSURE INDUCED APOPTOSIS SPHINGOSINE-1-PHOSPHATE ACTIVATION INHIBITOR AUTOPHAGY

发布时间 2024-05-25

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Molecular pharmacology.

2024年105卷3期

155-165页

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Fanning the Flames of Endoplasmic Reticulum (ER) Stress: Can Sphingolipid Metabolism Be Targeted to Enhance ER Stress-Associated Immunogenic Cell Death in Cancer?

Molecular pharmacology.

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Molecular pharmacology.

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Fanning the Flames of Endoplasmic Reticulum (ER) Stress: Can Sphingolipid Metabolism Be Targeted to Enhance ER Stress-Associated Immunogenic Cell Death in Cancer?

Molecular pharmacology.

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Molecular pharmacology.

相关期刊

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