首页> The Journal of biological chemistry> Oxidation of thymine to 5-formyluracil in DNA promotes misincorporation of dGMP and subsequent elongation of a mismatched primer terminus by DNA polymerase.

Oxidation of thymine to 5-formyluracil in DNA promotes misincorporation of dGMP and subsequent elongation of a mismatched primer terminus by DNA polymerase.

导出原文传递

翻译打印收藏纠错

作者： Masaoka A ^[1] ; Terato H ; Kobayashi M ; Ohyama Y ; Ide H

作者单位： Department of Mathematical and Life Sciences, Graduate School of Science, Hiroshima University, Higashi-Hiroshima 739-8526, Japan. ^[1]

关键词 Base Pair Mismatch DNA Polymerase I DNA Repair Deoxyguanine Nucleotides 碱基错配 DNA聚合酶Ⅰ DNA修复脱氧鸟嘌呤核苷酸类寡脱氧核糖核苷酸类胸腺嘧啶

发布时间 2013-11-20

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览0

The Journal of biological chemistry

2001年276卷19期

16501-16510页

相似文献

中文期刊
外文期刊
学位论文
会议论文

浏览0

点击下载本期目录封面

The Journal of biological chemistry

2001年276卷19期

16501-16510页

检索历史清除

Oxidation of thymine to 5-formyluracil in DNA promotes misincorporation of dGMP and subsequent elongation of a mismatched primer terminus by DNA polymerase.

The Journal of biological chemistry

相似文献

The Journal of biological chemistry

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Oxidation of thymine to 5-formyluracil in DNA promotes misincorporation of dGMP and subsequent elongation of a mismatched primer terminus by DNA polymerase.

The Journal of biological chemistry

相似文献

The Journal of biological chemistry

相关期刊

检索历史清除