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N-terminal deletion mutants of insulin-like growth factor-II (IGF-II) show Thr7 and Leu8 important for binding to insulin and IGF-I receptors and Leu8 critical for all IGF-II functions.

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作者： Hashimoto R ^[1] ; Fujiwara H ; Higashihashi N ; Enjoh Kimura T ; Terasawa H ; Fujita Yamaguchi Y ; Inagaki F ; Perdue JF ; Sakano K

作者单位： Molecular Biology Research Laboratory, Daiichi Pharmaceutical Co., Ltd., Tokyo, Japan. ^[1]

关键词 Insulin-Like Growth Factor II Leucine Receptor IGF Type 1 metabolism 胰岛素样生长因子Ⅱ 亮氨酸序列缺失苏氨酸

发布时间 2013-11-20

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The Journal of biological chemistry

1995年270卷30期

18013-18018页

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The Journal of biological chemistry

1995年270卷30期

18013-18018页

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N-terminal deletion mutants of insulin-like growth factor-II (IGF-II) show Thr7 and Leu8 important for binding to insulin and IGF-I receptors and Leu8 critical for all IGF-II functions.

The Journal of biological chemistry

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The Journal of biological chemistry

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N-terminal deletion mutants of insulin-like growth factor-II (IGF-II) show Thr7 and Leu8 important for binding to insulin and IGF-I receptors and Leu8 critical for all IGF-II functions.

The Journal of biological chemistry

相似文献

The Journal of biological chemistry

相关期刊

检索历史清除