XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease

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作者： Kaser A ^[1] ; Lee AH ^[2] ; Franke A ^[3] ; Glickman JN ^[4] ; Zeissig S ^[5] ; Tilg H ^[6] ; Nieuwenhuis EES ^[7] ; Higgins DE ^[8] ; Schreiber S ^[9] ; Glimcher LH

作者单位： Harvard Univ, Sch Publ Hlth, Dept Immunol & Infect Dis, Boston, MA 02115 USA ^[1] Innsbruck Med Univ, Dept Med, Christian Doppler Res Lab Gut Inflammat, A-6020 Innsbruck, Austria ^[2] Univ Kiel, Inst Clin Mol Biol, D-24105 Kiel, Germany ^[3] Univ Hosp Schleswig Holstein, Dept Med 1, D-24105 Kiel, Germany ^[4] Innsbruck Med Univ, Dept Med, Div Gastroenterol & Hepatol, A-6020 Innsbruck, Austria ^[5] Erasmus MC Sophia Childrens Hosp, Div Pediat Gastroenterol, NL-3000 GE Rotterdam, Netherlands ^[6] Harvard Univ, Sch Med, Dept Microbiol & Mol Genet, Boston, MA 02115 USA ^[7] Harvard Univ, Brigham & Womens Hosp, Sch Med, Dept Pathol, Boston, MA 02115 USA ^[8] Harvard Univ, Brigham & Womens Hosp, Sch Med, Div Gastroenterol,Dept Med, Boston, MA 02115 USA ^[9]

关键词 UNFOLDED PROTEIN RESPONSE PLASMA-CELL DIFFERENTIATION TRANSCRIPTION FACTOR XBP-1 CROHN-DISEASE ALPHA-DEFENSINS EPITHELIAL-CELLS PANETH CELLS HUMAN GENOME ASSOCIATION ACTIVATION

发布时间 2013-11-20

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