Lipidomics reveals the possible inhibition mechanism of light salting on the lipid degradation in grass carp(Ctenopharyngodon idella)muscle during extended cold storage
摘要Changes in lipids in light salt dry-curing(LSD)grass carp muscle stored at 4 ℃ for 15 days were investigated to clarify the effect of LSD on lipid transformation.A total of 1 265 lipid molecules from 35 subclasses were identified in the grass carp muscle.LSD promoted lipid conversion in early cold stage(0-6 days)but inhibited it later(6-15 days).Phosphatidylethanolamine(16:1e/22:6),phosphatidylcholine(16:0/20:4)and triacylglycerol(18:0/16:0/20:4)might be biomarkers of inhibited lipid corruption.The metabolisms of glycerophospholipid,fatty acids and arachidonic acid were crucial in restraining lipid transformations.Thiobarbituric acid reactive substances in LSD-pretreated muscle were significantly increased.LSD significantly increased acid lipase and phospholipase activities during early cold stage,but this effect decreased with refrigeration time.The lipid profile of LSD-pretreated grass carp muscle showed no significant change on day 15 of refrigeration.Consequently,LSD inhibited the lipid degradation of grass carp muscle during extended cold storage.
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