TNF-α impairs differentiation and function of TGF-β-induced Treg cells in autoimmune diseases through Akt and Smad3 signaling pathway

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作者单位 Institute of Health Sciences, Shanghai Institutes of Biological Sciences, Chinese Academy of Sciences and Shanghai JiaoTong University School of Medicine,Shanghai 200025, China ^[1] Department of Neuroimmunology, GlaxoSmithKline Research and Development Center, 898 Halei Road, Building 3, Zhangjiang Hi-Tech Park, Shanghai 201203,China ^[2] Institute of Health Sciences, Shanghai Institutes of Biological Sciences, Chinese Academy of Sciences and Shanghai JiaoTong University School of Medicine,Shanghai 200025, China;Department of Neuroimmunology, GlaxoSmithKline Research and Development Center, 898 Halei Road, Building 3, Zhangjiang Hi-Tech Park, Shanghai 201203,China ^[3]

关键词 tumor necrosis factor alpha (TNF-α),transforming growth factor beta (TGF-β),forkhead box P3 (Foxp3),Smad3,protein kinase B (Akt),autoimmune disease

栏目名称 Articles

DOI 10.1093/jmcb/mjs063

发布时间 2014-01-10

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分子细胞生物学报（英文版）

2013年5卷2期

85-98页

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TNF-α impairs differentiation and function of TGF-β-induced Treg cells in autoimmune diseases through Akt and Smad3 signaling pathway

分子细胞生物学报（英文版）

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分子细胞生物学报（英文版）

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TNF-α impairs differentiation and function of TGF-β-induced Treg cells in autoimmune diseases through Akt and Smad3 signaling pathway

分子细胞生物学报（英文版）

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分子细胞生物学报（英文版）

相关期刊

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