Regulation of adenosine triphosphate-sensitive potassium channels suppresses the toxic effects of amyloid-beta peptide (25-35)
关键词
neural regenerationneurodegenerative diseasesamyloid-β peptide (25–35)PC12 celladenosine triphosphate-sensitive potassium channelinducible nitric oxide synthasemitochondrial membrane potentialreactive oxygen speciesgrant-supported paperneur
DOI
10.3969/j.issn.1673-5374.2013.01.007
发布时间
2013-03-21(万方平台首次上网日期,不代表论文的发表时间)
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