The endoplasmic reticulum stress-autophagy pathway is involved in apelin-13-induced cardiomyocyte hypertrophy in vitro

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作者单位 Institute of Pharmacy and Pharmacology, Learning Key Laboratory for Pharmacoproteomics, University of South China, Hengyang 421001, China;Nanshan District Maternity & Child Healthcare Hospital of Shenzhen, Shenzhen 518052, China ^[1] Institute of Pharmacy and Pharmacology, Learning Key Laboratory for Pharmacoproteomics, University of South China, Hengyang 421001, China ^[2]

关键词 myocardial hypertrophy apelin APJ receptor ROS NOX4 ER stress autophagy reticulophagy

栏目名称 Cardiovascular Pharmacology

DOI 10.1038/aps.2017.97

发布时间 2018-01-03

基金项目

grants from the National Natural Science Foundation of China Hunan Provincial Natural Science Foundation China Postdoctoral Science Foundation (2014M560647).We especially thank Bing-bing WANG (Assistant Professor,Perinatal Biology Laboratory,Division of Maternal-Fetal Medicine,Rutgers University-Robert Wood Johnson Medical School,USA) for the English language correction

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

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中国药理学报（英文版）

2017年38卷12期

1589-1600页

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The endoplasmic reticulum stress-autophagy pathway is involved in apelin-13-induced cardiomyocyte hypertrophy in vitro

中国药理学报（英文版）

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中国药理学报（英文版）

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The endoplasmic reticulum stress-autophagy pathway is involved in apelin-13-induced cardiomyocyte hypertrophy in vitro

中国药理学报（英文版）

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中国药理学报（英文版）

相关期刊

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