Atypical chemokine receptor 3 induces colorectal tumorigenesis in mice by promoting β-arrestin-NOLC1-fibrillarin-dependent rRNA biogenesis

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作者 Juan Yang ^[1] Rong-rong Miao ^[2] Ya-nan Li ^[3] Ting Pan ^[3] Shu-hua Wu ^[4] Xian-jun Qu ^[2] Shu-xiang Cui ^[3] 学术成果认领

作者单位 Beijing Key Laboratory of Environmental Toxicology,Department of Toxicology and Sanitary Chemistry,School of Public Health,Capital Medical University,Beijing 100069,China;Department of Comprehensive Ward,Affiliated Hospital of Guizhou Medical University,Guiyang 550004,China ^[1] Department of Pharmacology,School of Basic Medical Sciences,Capital Medical University,Beijing 100069,China ^[2] Beijing Key Laboratory of Environmental Toxicology,Department of Toxicology and Sanitary Chemistry,School of Public Health,Capital Medical University,Beijing 100069,China ^[3] Department of Pathology,Hospital of Bin Zhou Medical College,Binzhou 256603,China ^[4]

关键词 tumorigenesis ACKR3 NOLC1 fibrillarin ribosome biogenesis Villin-ACKR3 transgenic mice

栏目名称 Chemotherapy

发布时间 2022-12-16

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中国药理学报（英文版）

2022年43卷11期

2967-2976页

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Atypical chemokine receptor 3 induces colorectal tumorigenesis in mice by promoting β-arrestin-NOLC1-fibrillarin-dependent rRNA biogenesis

中国药理学报（英文版）

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中国药理学报（英文版）

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Atypical chemokine receptor 3 induces colorectal tumorigenesis in mice by promoting β-arrestin-NOLC1-fibrillarin-dependent rRNA biogenesis

中国药理学报（英文版）

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中国药理学报（英文版）

相关期刊

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