摘要Background:Chronic subjective tinnitus affects 15-20％of adults globally,with 3-5％experiencing severe quality-of-life impairment.Sound therapy(ST)is a core intervention,but its neuromodulatory mechanisms remain incompletely characterized due to the lack of objective biomarkers.Meth-ods:Following PRISMA guidelines,we systematically searched PubMed,Web of Science,Embase,and other databases(1977-2025)for randomized controlled trials(RCTs)investigating ST effects on EEG spectral power in tinnitus patients.Inclusion criteria required pre/post-ST EEG data in delta(0.5-4 Hz)and alpha(8-14 Hz)bands.Two independent reviewers extracted data and assessed bias using Cochrane tools.Standardized mean differences(SMDs)with 95％confidence intervals(CIs)were pooled via fixed/random-effects models.Results:Two RCTs(n=71 patients)met inclusion criteria.Post-ST,the intervention group showed non-significant trends toward increased delta(SMD=0.11,95％CI:[-0.40,0.62],P=0.67)and alpha(SMD=0.09,95％CI:[-0.41,0.60],P=0.72)power.Between-group analyses revealed greater alpha enhancement in customized ST versus controls(SMD=0.40,95％CI:[-0.12,0.91],P=0.13;I2=39％).Alpha changes demonstrated moderate consistency across studies(I2=39％,P=0.19).Con-clusion:ST induces measurable delta/alpha power modulation,suggesting enhanced inhibitory neurotransmission and attentional regulation.While statistical significance was limited by sample size,these trends support EEG as a tool for objective treatment monitoring.We propose a"dual-band synergistic neuromodulation"framework:ST concurrently suppresses pathological hyperexcitability and potentiates slow-wave inhibition.Standardized EEG protocols and longitudinal validation are critical for advancing precision tinnitus therapeutics.