摘要Anxiety disorder is a common and serious mental disorder. At present, the pathogenesis of anxiety disorder includes hypothalamus-pituitary-adrenal (HPA) axis, neuroimmune, and brain-gut axis disorders, among others. This paper discusses the correlation between anx-iety disorder and the hypothalamus-pituitary-endocrine axis and finds that immune inflammation can be used as a "bridge" between the hypothalamus-pituitary-endocrine axis and anxiety disorder.
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